In this area you will be able to:
- Propose, vote on, and discuss research ideas
- View current studies
- View published research
Here, you can submit a research idea to the community, cast your votes, and discuss research ideas proposed by other members. Please make your research question as specific as possible. Other members will vote on your research idea, and we will prioritize research ideas with the most votes.
You are allowed to vote for your own proposed research idea if you want. However, you can only vote for a total of five research ideas. If you have already cast your five votes and an idea you like even more is proposed, you can change your votes at any time to reflect your current preferences.
The research team will review all submitted ideas and provide a response to you and to the community. If your idea leads to an IBD Partners Study, you will have the opportunity to serve as a patient collaborator on the research team for that study.
We encourage you to prioritize the ideas that are most important to you, even if the research team determines that your idea is not a good fit for IBD Partners. We will share ideas labeled “Not a Good Fit” with researchers outside of our network when appropriate. We want to make sure all of your votes count!
Thanks for your participation in this important platform to help the IBD research community understand what research questions are important to patients. We are passionate about finding answers to your questions!
What is the difference between diseased/inflamed tissue in Crohn's or UC and the seemingly healthy tissue WITHIN THE SAME PERSON?
This is important because in many cases of IBD, there is healthy tissue. I had UC covering only half my colon. Why was the other half of it healthy? What was going on within that tissue that left it healthy and intact rather than inflamed? Why does UC spread over time if uncontrolled? Why isn't the whole colon/intestinal tract inflamed all the time?
I have UC and waiting to hear if I now have Graves Disease.
It is important to research various anti-inflammatory options.
Is IBD/flaring commonly associated with gynecologic manifestations, such as lichen sclerosus or localized inflammation at the labia/vaginal vault?
There's a lot of information available about some specific extra-intestinal manifestations, but not in this area. More than 50% of the IBD Partners participants are female, and studies already document a correlation between hormones and flaring.
Can reducing inflammation and increasing gut health through the removal of parasites improve ulcerative colitis?
I've personally have experienced reduced inflammation from removal of parasites. A great video series that is currently showing for free is https://autoimmunesecrets.com. The end of video 4 mentioned the following herbs for parasite removal: * mimosa pudica * neem * clove * vidanga (an Ayurvedic herb)
What approaches to healing did people diagnosed with IBD and experiencing long term "remission" claim were most effective in achieving this outcome?
I think it's important to study people who've for all practical purposes recovered from IBD. I've come across many people like this. I'd like to know more about what they did.
Are the methods Donna Jackson Nakasawa (see: https://donnajacksonnakazawa.com/) proposes to help people heal themselves of auto-immune illness effective for IBD?
Donna has proposed in her book "Childhood Disrupted" a number of methods for healing the effects of Adverse Childhood Experiences (ACES). She claims the correlation between ACEs and autoimmune illness (particularly for women) later on in life is very strong (https://www.cdc.gov/violenceprevention/acestudy/).
In 2014 I went into A-FIB because I was deprived of vitamins and nutrients in my system. Since then I am taking very powerful supplements that work! Does this happen to many Crohns patients?
I was taking many supplements and nutrients at the time. I had no warning signs that this could happen. P.S. the cardiologist said my heart was healthy. It's important for patients to know, that proper nutrition is a must!
Rat models of IBD usually can't get into this because of the way they induce colitis is different than how humans get UC. Why does UC generally start in the rectum and move its way upward? What causes it to often start there and then what causes it to spread?
Diet Low in Red and Processed Meat Does Not Reduce Rate of Crohn’s Disease Flares
Impact of Obesity on Disease Activity andPatient-Reported Outcomes Measurement InformationSystem (PROMIS) in Inflammatory Bowel Diseases